Can scientists leverage mysterious mossy cells for brain disease treatments?

A small population of brain cells deep in a memory-making region of the brain controls the production of new neurons ...
read more

Our article in Neurological Sciences

A review and meta-analysis of stem cell therapies in stroke patients: effectiveness and safety evaluation...
read more

Our co-authored article at Neurochemistry International

We would like to inform you, that our co-authored article ...
read more

Our first video back in 2018…

5

JOURNAL ARTICLES

50

THOUSAND USD GRANTS RECEIVED

5

RESEARCH MEMBERS

Currently working on stroke research

We are working on the development of animal models of stroke to understand the intrinsic activation of brain defense mechanisms towards injury. For this we developed a close loop deep brain stimulation system to test the role of striatum stimulation on the behavioural changes after ischemic stroke.

  • Main findings:

    Striatum stimulation alleviates post stroke tissue
    damage.
  • Striatum stimulation suppresses contralateral hemispheric activity
  • Animals, who received stimulation, outperform control group in behavioural tests
View all Publications

A review and meta-analysis of stem cell therapies in stroke patients: effectiveness and safety evaluation

L Hovhannisyan, S Khachatryan, A Khamperyan , S Matinyan

Ischemia-Induced Neuronal Changes May Be Guided By Physical Activity

Matinyan S.V., Hayrapetyan L.M., Ghandilyan E.A., Anastasyan R.A., Khachatryan V.V., Harutyunyan T.A, Minasyan E.A., Khanperyan A.Kh.

Alzheimer’s disease-like pathology-triggered oxidative stress, alterations in monoamines levels, and structural damage of locus coeruleus neurons are partially recovered by a mix of proteoglycans of embryonic genesis

Aghajanov M, Chavushyan V, Matinyan S, Danielyan M, Yenkoyan K.

Embryonic proteoglycans regulate monoamines in the rat frontal cortex and hippocampus in Alzheimer’s disease-like pathology

Embryonic proteoglycans regulate monoamines in the rat frontal cortex and hippocampus in Alzheimer's disease-like pathology.
Publisher's website
> View All
View All